Mini Symposium 2026 Apr 8 Andreas Nicolaides: Difference between revisions

Mini Symposium 2026 Apr 8 Andreas Nicolaides (view source)

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 # Stabilisers: systems that preserve lineage coherence, such as centromeric divergence with drive suppression, piRNA surveillance, inversions, supergenes, imprinting, and incompatibility complexes.
- These functions do not direct development itself but transform the regulatory logic that structures it. In this sense, the system constitutes a meta-programme: a higher-order genomic architecture that converts existing developmental programmes into novel ones, linking organisms across space and time. Crucially, they also resolve the anomalies: stasis reflects stability maintained by stabilisers, sudden transformations occur when initiators cross thresholds, reticulated histories arise from coordinating processes across lineages, and hybrid dysfunction stems from divergence in stabilising systems.
+These functions do not direct development itself but transform the regulatory logic that structures it. In this sense, the system constitutes a meta-programme: a higher-order genomic architecture that converts existing developmental programmes into novel ones, linking organisms across space and time. Crucially, they also resolve the anomalies: stasis reflects stability maintained by stabilisers, sudden transformations occur when initiators cross thresholds, reticulated histories arise from coordinating processes across lineages, and hybrid dysfunction stems from divergence in stabilising systems.
- On this basis, the hypothesis yields distinctive predictions: genomic turnover should track clade-specific tempos of speciation; bursts of mobile elements and duplications should cluster around radiation events; shared viral or symbiotic agents should generate concordant genomic change; and hybrid dysfunction should correlate with divergence in coherence-preserving systems. Evolutionary anomalies, on this view, are not noise but signatures of a genomic meta-programme in action.
+On this basis, the hypothesis yields distinctive predictions: genomic turnover should track clade-specific tempos of speciation; bursts of mobile elements and duplications should cluster around radiation events; shared viral or symbiotic agents should generate concordant genomic change; and hybrid dysfunction should correlate with divergence in coherence-preserving systems. Evolutionary anomalies, on this view, are not noise but signatures of a genomic meta-programme in action.